Prof Dr Jan Glatz (J.F.C.)

Wong, L. Y., Torfs, T., Vanherle, S. J. V., Janssen, J., Claes, G. R. F., Stroeks, S. L. V. M., Willemars, M. M. A., Schianchi, F., Kapsokalyvas, D., Weltjens, E., Swinnen, A., Strzelecka, A., Krapels, I. P. C., Heymans, S. R. B., Glatz, J., van den Wijngaard, A., Brunner, H. G., Broers, J., Luiken, J. F. P., ... Nabben, M. (2025). Clinical and metabolic consequences of a historic pathogenic lamin A/C founder variant. Scientific Reports, 15(1), Article 23842. https://doi.org/10.1038/s41598-025-08495-0

Papur, O. S., Glatz, J. F. C., & Luiken, J. J. F. P. (2024). Protein kinase-D1 and downstream signaling mechanisms involved in GLUT4 translocation in cardiac muscle. Biochimica et biophysica acta. Molecular cell research, 1871(6), Article 119748. https://doi.org/10.1016/j.bbamcr.2024.119748

Wang, F., Neumann, D., Kapsokalyvas, D., Hoes, M. F., Schianchi, F., Glatz, J. F. C., Nabben, M., & Luiken, J. J. F. P. (2024). Specific Compounds Derived from Traditional Chinese Medicine Ameliorate Lipid-Induced Contractile Dysfunction in Cardiomyocytes. International Journal of Molecular Sciences, 25(15), Article 8131. https://doi.org/10.3390/ijms25158131

Glatz, J. F. C., Heather, L. C., & Luiken, J. J. F. P. (2024). CD36 as a gatekeeper of myocardial lipid metabolism and therapeutic target for metabolic disease. Physiological reviews, 104(2), 727-764. https://doi.org/10.1152/physrev.00011.2023

Wang, S., Han, Y., Liu, R., Hou, M., Neumann, D., Zhang, J., Wang, F., Li, Y., Zhao, X., Schianchi, F., Dai, C., Liu, L., Nabben, M., Glatz, J. F. C., Wu, X., Lu, X., Li, X., & Luiken, J. J. F. P. (2024). Glycolysis-Mediated Activation of v-ATPase by Nicotinamide Mononucleotide Ameliorates Lipid-Induced Cardiomyopathy by Repressing the CD36-TLR4 Axis. Circulation Research, 134(5), 505-525. https://doi.org/10.1161/CIRCRESAHA.123.322910

Wang, S., Neumann, D., Westenbrink, B. D., Schianchi, F., Wong, L.-Y., Sun, A., Strzelecka, A., Glatz, J. F. C., Luiken, J. J. F. P., & Nabben, M. (2022). Ketone Body Exposure of Cardiomyocytes Impairs Insulin Sensitivity and Contractile Function through Vacuolar-Type H+-ATPase Disassembly-Rescue by Specific Amino Acid Supplementation. International journal of molecular sciences, 23(21), Article 12909. https://doi.org/10.3390/ijms232112909

Wang, S. J., Han, Y. Y., Nabben, M., Neumann, D., Luiken, J. J. F. P., & Glatz, J. F. C. (2022). Endosomal v-ATPase as a Sensor Determining Myocardial Substrate Preference. Metabolites, 12(7), Article 579. https://doi.org/10.3390/metabo12070579

Glatz, J. F. C., Nabben, M., & Luiken, J. J. F. P. (2022). CD36 (SR-B2) as master regulator of cellular fatty acid homeostasis. Current Opinion in Lipidology, 33(2), 103-111. https://doi.org/10.1097/MOL.0000000000000819

Glatz, J., Wang, F., Nabben, M., & Luiken, J. (2022). Myocardial lipid metabolism: Targeting CD36 to treat cardiac disease. Research Outreach, (128), 86-89. https://researchoutreach.org/publications/issue-128/

Wang, S. J., Schianchi, F., Neumann, D., Wong, L. Y., Sun, A. M., van Nieuwenhoven, F. A., Zeegers, M. P., Strzelecka, A., Col, U., Glatz, J. F. C., Nabben, M., & Luiken, J. J. F. P. (2021). Specific amino acid supplementation rescues the heart from lipid overload-induced insulin resistance and contractile dysfunction by targeting the endosomal mTOR-v-ATPase axis. Molecular Metabolism, 53, Article 101293. https://doi.org/10.1016/j.molmet.2021.101293